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  [11]美国NKF-K/DOQI工作组.慢性肾脏病及透析的临床实践指南[M].北京:人民卫生出版社,2003(1).

  [12]郭义敏,刘颖,陈历明.近端肾小管碳酸氢根重吸收的分子机制及代谢性酸中毒[J].生理学报,2014,66(4): 398~414.

  [13]Moon, et al. The Intrinsic Antimicrobial Activity of Bamboo Salt against Salmonella enteritidis[J]. Molecular and Cellular Toxicology, 2010,6(1): 109-109.

  医学论文参考文献

  [1]Weaver A., da Silva A. G, Nuttall R. K.,et al. An elevated matrix metalloproteinase(MMP) in an animal model of multiple sclerosis is protective by affecting Thl/Th2polarization. The FASEB journal. 2005. 19(12): p. 1668-1670.

  [2]Venteo L_,Bourlet T” Renois F” et al. Enterovirus-related activation of thecardiomyocyte mitochondrial apoptotic pathway in patients with acute myocarditis.European heart journal. 2010. 31(6): p. 728-736.

  [3]Rose N. R. The adjuvant effect in infection and autoimmunity. Clinical reviews inallergy & immunology. 2008. 34(3): p. 279-282.

  [4]陈瑞珍,解玉泉.病毒性心肌炎向扩张型心肌病演变的免疫学机制.诊断学理论与实践-2012. 10(5): p. 414-417.

  [5]Camporeale A., Marino F” Papageorgiou A.,et al. STAT3 activity is necessary andsufficient for the development of immune “ mediated myocarditis in mice andpromotes progression to dilated cardiomyopathy. EMBO molecular medicine. 2013.5(4): p. 572-590.

  [6]Zhu J.,Paul W. E. CD4 T cells: fates, functions, and faults. Blood. 2008. 112(5): p.1557-1569.

  [7]Korn T.,Bettelli E?,Oukka M.,et al. IL-17 and Thl7 Cells. Annual review ofimmunology. 2009. 27: p. 485-517.

  [8]Yu Y” Liu Y-,Shi F.-D., et al. Cutting edge: leptin-induced RORyt expression in CD4+T cells promotes Thl7 responses in systemic lupus erythematosus. The Journal ofImmunology. 2013. 190(7): p. 3054-3058.

  [9]van Bruggen N.,Ouyang W. Thl7 Cells at the Crossroads of Autoimmunity,Inflammation, and Atherosclerosis. Immunity. 2014. 40(1): p. 10-12.

  [10]Annnn2:iatoF., Cosmi L., Santarlasci V.,et al. Phenotypic and functional features ofhuman Thl7 cells. The Journal of experimental medicine. 2007. 204(8): p. 1849-1861.

  [11]ShahraraS.,Huang Q.,Mandelin II A. M.,et al. TH-17 cells in rheumatoid arthritis.Arthritis Research and Therapy. 2008. 10(4): p. R93.

  [12]KomiyamaY.,Nakae S.,Matsuki T_,et al. IL-17 plays an important role in thedevelopment of experimental autoimmune encephalomyelitis. The Journal ofImmunology. 2006. 177(1): p. 566-573.

  [13]Bettelli E” Carrier Y?,Gao W,et al. Reciprocal developmental pathways for thegeneration of pathogenic effector TH17 and regulatory T cells. Nature. 2006.441(7090): p. 235-238.

  [14]VeldhoenM.,Hocking R. J.,Atkins C. J.,et al. TGFp in the context of aninflammatory cytokine milieu supports de novo differentiation of IL-17-producing Tcells. Immunity. 2006. 24(2): p. 179-189.

  [15]ManganP. R., Harrington L. E.,OQuinn D. B.,et al. Transforming growth factor-pinduces development of the TH17 lineage. Nature. 2006. 441(7090): p. 231-234.

  [16]AkimzhanovA. M.,Yang X. 0.,Dong C. Chromatin remodeling of interleukin-17(IL-17)-IL-17F cytokine gene locus during inflammatory helper T cell differentiation.Journal of biological chemistry. 2007. 282(9): p. 5969-5972.

  [17]NurievaR” Yang X. 0” Martinez G,et al. Essential autocrine regulation by IL-21 inthe generation of inflammatory T cells. Nature. 2007. 448(7152): p. 480-483.

  [18]Yang L,Anderson D. E.,Baecher-Allan C.,et al. IL-21 and TGF-&bgr; are requiredfor differentiation of human TH17 cells. Nature. 2008.454(7202): p. 350-352.

  [19]TrinchieriG,Pflanz S.,Kastelein R. A. The IL-12 family of heterodimeric cytokines:new players in the regulation of T cell responses. Immunity. 2003. 19(5): p. 641-644.

  [20]Zhou L,Ivanov I. I,Spolski R?,et al. IL?6 programs TH-17 cell differentiation bypromoting sequential engagement of the IL-21 and IL-23 pathways. Natureimmunology. 2007. 8(9): p. 967-974.

  [21]AggarwalS” Ghilardi N., Xie M.-H.,et al. Interleukin-23 promotes a distinct CD4 Tcell activation state characterized by the production of interleukin-17. Journal ofBiological Chemistry. 2003. 278(3): p. 1910-1914.

  [22]Harrington L. E., Hatton R. D.,Mangan P. R.,et al. Interleukin 17—producing CD4+effector T cells develop via a lineage distinct from the T helper type 1 and 2 lineages.Nature immunology. 2005. 6(11): p. 1123-1132.

  [23]Yang X. 0., Panopoulos A. D.,Nurieva R.,et al. STAT3 regulates cytokine-medlatedgeneration of inflammatory helper T cells. Journal of Biological Chemistry. 2007.282(13): p. 9358-9363.

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