返回
首页 > 参考文献
3 页 客服微信号: bylw8com 客服QQ:3346581880

  [24]LaurenceA.,Tato C. M.,Davidson T. S?,et al. Interleukin-2 signaling via STAT5constrains T helper 17 cell generation. Immunity. 2007. 26(3): p. 371-381.

  [25]EberlG,Marmon S.,Sunshine M.-J.,et al. An essential function for the nuclearreceptor RORyt in the generation of fetal lymphoid tissue inducer cells. Natureimmunology. 2004. 5(1): p. 64-73.

  [26]Yang X. 0” Pappu B. P.,Nurieva R., et al. T helper 17 lineage differentiation isprogrammed by orphan nuclear receptors RORa and RORy. Immunity. 2008. 28(1): p.29-39.

  [27]IvanovI. I.,McKenzie B. S.,Zhou L.,et al. The Orphan Nuclear Receptor RORytDirects the Differentiation Program of Proinflammatory IL-17< sup>+ T HelperCells. Cell. 2006. 126(6): p. 1121-1133.

  [28]QingK.,Weifeng W” Fan Y?,et al. Distinct different expression of Thl7 and Th9 cellsin coxsackie virus B3-induced mice viral myocarditis. Wol J. 2011. 8: p. 267.

  [29]Jiang N.,Liao W”Kuang X. Effects of emodin on IL-23/IL-17 inflammatory axis,Thl7 cells and viral replication in mice with viral myocarditis. Nan Fang Y Ke DaXue Xue Bao. 2014. 34(3): p. 373-378.

  [30]Yang F.,Wu W.-F.,Yan Y.-L.,et al. Expression of il-23/thl7 pathway in a murinemodel of coxsackie virus b3-induced viral myocarditis. Virology journal. 2011. 8(1): p.301.

  [31]FanY” Weifeng W., Yuluan Y” et al. Treatment with a neutralizing anti-murineinterleukin-17 antibody after the onset of coxsackievirus b3-induced viral myocarditisreduces myocardium inflammation. Virol J. 2011. 8(1): p.17.

  [32]Yu M.5 Hu J” Zhu M. X.,et al. Cardiac fibroblasts recruit Thl7 cells infiltration intomyocardium by secreting CCL20 in CVB3-induced acute viral myocarditis. CellPhysiol Biochem. 2013. 32(5): p, 1437-1450.

  [33]XieY.,Chen R.,Zhang X.,et al. The role of Thl7 cells and regulatory T cells inCoxsackievirus B3-induced myocarditis. Virology. 2011. 421(1): p. 78-84.

  [34]YuanJ., Yu M., Lin Q.-W., et al. Thl7 cells contribute to viral replication incoxsackievirus B3-induced acute viral myocarditis. The Journal of Immunology. 2010.185(7): p. 4004-4010.

  [35]YuanJ.,Cao A.-L., Yu M.,et al. Thl7 cells facilitate the humoral immune response inpatients with acute viral myocarditis. Journal of clinical immunology. 2010. 30(2): p.226-234.

  [36]Yuan J” Yu M.,Lin Q.-W., et al. Neutralization of IL-17 inhibits the production ofanti-ANT autoantibodies in CVB3-induced acute viral myocarditis. Internationalimmunopharmacology. 2010.10(3): p. 272-276.

  [37]Liu Y?,Zhu H?,Su Z.,et al. IL-17 contributes to cardiac fibrosis followingexperimental autoimmune myocarditis by a PKCbeta/Erkl/2/NF-kappaB_dependentsignaling pathway. Int Immunol. 2012. 24(10): p. 605-612.

  [38]BaldevianoG C., Barin J. G,Talor M. V” et al. Interleukin-17A is dispensable formyocarditis but essential for the progression to dilated cardiomyopathy. Circulationresearch. 2010. 106(10): p. 1646-1655.


首页 上一页 1 2 3

猜你喜欢

版权所有 Copyright©2006-2024 毕业论文网 版权所有

苏ICP备14005682号

联系邮箱:Lw54@vip.qq.com